Dermatology - Pitryiasis Versicolor
Malassezia furfur is a lipophilic yeast that generally dwells in the keratin of skin and hair follicles of humans who have reached adolescence and beyond. Pityriasis versicolor, which is also known as tinea versicolor in the United States, is linked to the superficial overgrowth of Malassezia furfur. In the context of hyperhidrosis and moist skin, this infection does not spread from person to person; rather, it is caused by an excess of resident cutaneous flora, also known as the cutaneous microbiome. Despite the fact that lesions are typically asymptomatic, patients may be concerned about dyspigmentation due to its cosmetic effects. Lesions that have been present for a number of months or years. lesions (plural) In addition to having a sharp margin, macules can be round or oval in shape, pink to brown in color, and changing in size. Macules also have fine scaling, which can be fully understood by gently abrading lesions. Scale is absent in lesions that have been treated or resolved. Expanding and merging lesions can result in the formation of huge geographic areas. In skin that has been tanned or dyed, lesions appear white or off-white in color. Direct microscopic examination with potassium hydroxide (KOH) is the method that was used to make the diagnosis. Vitiligo, pityriasis alba, postinflammatory hypopigmentation, tinea corporis, seborrheic dermatitis, and cutaneous T cell lymphoma are all included in the differential diagnosis. Selenium sulfide lotion or shampoo at a concentration of 2.5%, as well as ketoconazole shampoo, azole creams (including ketoconazole, econazole, micronazole, and clotrimazole), or terbinafine solution at a concentration of 1% are examples of topical agents. The administration of 400 milligrams of either itraconazole, fluconazole, or ketoconazole is included in the systemic therapy.
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