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Emergency and Acute Medicine: Thoracic Spine Injury




Thoracic spine injuries are typically the result of high-energy trauma, as the thoracic region is relatively rigid due to stabilization by the rib cage and costovertebral joints. Common mechanisms include axial compression, flexion–rotation, shear forces, flexion–distraction, and extension. Because of this rigidity, a large force is usually required to produce fractures or dislocations, most commonly seen in motor vehicle collisions, falls from significant heights, or high-impact trauma. The thoracic spinal canal is also relatively narrow, increasing the risk of neurologic injury when fractures occur—especially near the thoracolumbar junction (T11–L2), where a large proportion of spinal injuries are concentrated.


The stability of the thoracic spine is best understood using the three-column model. The anterior column includes the anterior vertebral body and supporting ligaments, the middle column consists of the posterior vertebral body and posterior longitudinal ligament, and the posterior column includes the vertebral arch and ligamentous structures. Injury involving two or more columns indicates instability, which significantly increases the risk of neurologic compromise.


Thoracic spine fractures are broadly categorized into minor and major injuries. Minor fractures include isolated spinous process, transverse process, or articular fractures, which are generally stable. Major injuries include compression fractures, burst fractures, seatbelt (flexion–distraction) injuries, and fracture-dislocations. Compression fractures typically involve the anterior column and may remain stable if the middle column is intact. In contrast, burst fractures involve the middle column and may result in retropulsion of bone fragments into the spinal canal, posing a risk of spinal cord compression. Flexion–distraction injuries, often associated with improper seatbelt use, disrupt posterior and middle columns, while fracture-dislocations represent complete failure of all three columns and are highly unstable with a high likelihood of neurologic injury.


Clinically, patients usually present with localized thoracic back pain, tenderness, bruising, or deformity, such as step-offs or widened interspinous spaces. Pain may also be referred to the chest or abdomen. Because of the force required to injure the thoracic spine, associated internal injuries should always be suspected, including thoracic, abdominal, or vascular trauma. Neurologic findings may include paresthesia, weakness, loss of reflexes, bowel or bladder dysfunction, or signs of spinal shock such as hypotension with bradycardia. The presence of injury at another spinal level should increase suspicion for thoracic involvement.


Evaluation begins with primary trauma assessment (ABCs) followed by a detailed neurologic and spinal examination, including rectal tone and perianal sensation. Any patient with midline tenderness, altered mental status, intoxication, distracting injuries, or significant mechanism of injury should undergo imaging. Initial imaging typically includes anteroposterior and lateral radiographs, but CT scanning is more sensitive and is often required for definitive assessment. CT is particularly useful for identifying fractures and evaluating stability, while MRI is indicated when there is concern for spinal cord injury, ligamentous damage, or neurologic deficits. Importantly, identification of a fracture in one region of the spine mandates imaging of the entire spine, as multiple injuries are not uncommon.


Management requires strict spinal immobilization throughout evaluation and resuscitation. Airway management should be performed with in-line stabilization, and hypotension should initially be assumed to result from hemorrhage until proven otherwise. Neurogenic shock, characterized by hypotension with bradycardia and warm extremities, should be distinguished from hypovolemic shock and may require vasopressor support in addition to fluid resuscitation.


Patients with suspected spinal cord injury or unstable fractures require urgent consultation with neurosurgical or orthopedic spine specialists and should be managed in a trauma center. Pain control with opioids, NSAIDs, and adjuncts is essential. The use of high-dose corticosteroids remains controversial and is no longer routinely recommended, though it may be considered in select cases within a limited time window after injury.


Admission is indicated for patients with unstable injuries, neurologic deficits, significant pain, ileus, or associated trauma. ICU care may be required depending on severity. Selected patients with stable minor fractures and no neurologic impairment may be discharged after specialist evaluation, with close outpatient follow-up.


Key pitfalls include failure to recognize thoracic spine injury in patients with major trauma, underuse of CT imaging in high-risk cases, and premature removal of spinal precautions. Maintaining immobilization until injury is definitively excluded, performing thorough neurologic assessments, and involving specialists early are critical steps in preventing missed injuries and improving patient outcomes.

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