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Orthopaedic Surgery - Peroneal Tendon Subluxation
Basics
Peroneal tendon subluxation is abnormal displacement of the peroneus longus and/or peroneus brevis tendons from their normal groove behind the lateral malleolus.
The two principal peroneal tendons are:
Peroneus longus
and
Peroneus brevis.
An accessory muscle, the:
Peroneus quartus
is variably present and has been reported in up to approximately 20% of individuals.
Anatomy
The peroneus longus and brevis pass posterior to the:
Lateral malleolus
within a common tenosynovial sheath.
They are stabilized by the:
Superior peroneal retinaculum
and more distally by the:
Inferior peroneal retinaculum.
The superior peroneal retinaculum is particularly important in maintaining the tendons within the:
Retromalleolar groove of the distal fibula.
Subluxation and Dislocation
When the superior peroneal retinaculum is torn, stretched, or developmentally insufficient, the tendons may translate:
Anteriorly over the lateral malleolus
during ankle motion.
This may produce:
Pain
Snapping
Clicking
or a:
Palpable tendon displacement.
The instability may be:
Acute after trauma
or
Chronic and recurrent.
Epidemiology
Peroneal tendon instability is:
Uncommon.
It is encountered most frequently in:
Active adolescents and adults, especially athletes.
Risk Factors
Athletic activities that place substantial stress on the lateral ankle increase risk.
Other predisposing factors include:
Generalized ligamentous laxity
Shallow or convex retromalleolar groove
Chronic ankle instability
Cavovarus or varus hindfoot alignment
Accessory peroneal musculature
Genetics
There is no established Mendelian inheritance pattern.
However, inherited anatomy such as:
Ligamentous laxity
or
Hindfoot alignment
may contribute indirectly.
Etiology
The classic acute mechanism involves:
Forceful ankle dorsiflexion
combined with:
Eversion
and strong contraction of the:
Peroneal muscles.
This can tear or strip the:
Superior peroneal retinaculum
from the distal fibula.
Sports Association
The injury is classically associated with:
Skiing
but may also occur in:
Running
Soccer
Basketball
Football
Dance
and other activities involving abrupt ankle loading.
Chronic or Spontaneous Instability
Some patients develop instability without a major traumatic event.
Predisposing anatomic factors may include:
A shallow fibular groove
Generalized ligamentous laxity
Varus hindfoot
Abnormal peroneal anatomy
Associated Conditions
Peroneal tendon subluxation may coexist with:
Chronic lateral ankle instability
Peroneus brevis split tear
Peroneal tenosynovitis
Cavovarus alignment
Generalized ligamentous laxity
Diagnosis
Diagnosis is based largely on:
History
Dynamic physical examination
and, when needed,
Imaging.
Because the tendons may be normally positioned at rest, dynamic evaluation is particularly important.
Signs and Symptoms
Patients commonly report:
Persistent posterolateral ankle pain
Pain with activity
Snapping or popping behind the fibula
A sensation of the tendons moving over the lateral malleolus
History
A typical history may involve:
A skier or running athlete
with:
Painful lateral ankle snapping
following an inversion/eversion-type injury or forceful dorsiflexion.
Patients may also report:
Repeated ankle sprains
or
Chronic instability.
Acute Presentation
Following an acute injury, there may be:
Swelling
Ecchymosis
Tenderness posterior to the lateral malleolus
Chronic Presentation
Chronic cases may have relatively little swelling but demonstrate:
Reproducible painful snapping
during active ankle movement.
Physical Examination
Inspection
Inspect the posterolateral ankle for:
Swelling
Bruising
Visible tendon displacement
Hindfoot alignment
Neurovascular Examination
Assess:
Distal pulses
Sensation
Motor function
because lateral ankle trauma can occasionally involve nearby neurologic structures.
Ankle Stability
Evaluate the lateral ligament complex using:
Anterior drawer testing
and
Talar tilt or inversion stress testing.
This is important because chronic peroneal instability may coexist with:
Lateral ligament instability.
Provocative Maneuver
The patient can be asked to move from:
Plantarflexion and eversion
into:
Dorsiflexion with active eversion.
This contracts the peroneal muscles and may reproduce:
Posterolateral pain
Snapping
or
Visible/palpable tendon subluxation.
Palpation
The examiner should palpate directly:
Behind the lateral malleolus
while the patient repeatedly:
Inverts and everts the foot
or performs resisted ankle motion.
The tendons may be felt moving:
Anteriorly over the fibula.
Normal Tendon Clicking
Peroneal tendons may occasionally:
Snap or click within their sheath
without actual instability.
Therefore, the finding is clinically meaningful when there is:
True displacement from the groove
together with:
Reproduction of the patient’s pain or symptoms.
Laboratory Tests
There are no specific laboratory tests for isolated peroneal tendon subluxation.
Imaging
Plain Radiographs
Initial ankle radiographs help assess for:
Fracture
Bony avulsion
Hindfoot alignment
A small avulsion fragment from the posterolateral distal fibula may indicate injury to the:
Superior peroneal retinaculum.
Historically, such avulsion fractures have been reported in approximately:
10% of acute cases.
CT
CT can define:
The shape and depth of the retromalleolar groove
and other bony anatomy.
It may be helpful for:
Preoperative planning
when groove morphology is abnormal.
MRI
MRI can demonstrate:
Superior peroneal retinacular tearing or attenuation
Peroneal tenosynovitis
Fluid within the tendon sheath
Peroneus brevis longitudinal split tear
Associated ligamentous injuries
MRI is also useful for identifying chronic tendon degeneration.
Dynamic Ultrasound
Dynamic musculoskeletal ultrasound is particularly useful because the tendons can be observed:
During active ankle movement.
It may directly demonstrate:
Subluxation or dislocation over the lateral malleolus.
Because instability may not be present at rest, dynamic ultrasound can sometimes be more informative than static imaging.
Diagnostic Injection
A local anesthetic injection into the:
Peroneal tendon sheath
may occasionally help determine whether pain originates from the peroneal tendons.
This can be useful when the diagnosis remains uncertain.
Pathological Findings
Possible findings include:
Shallow retromalleolar groove
Torn or attenuated superior peroneal retinaculum
Peroneal tenosynovitis
Longitudinal split tear of the peroneus brevis
Chronic Instability
Repeated subluxation can cause friction and progressive damage to the:
Peroneus brevis tendon, particularly a longitudinal split tear.
Differential Diagnosis
Important alternative diagnoses include:
Lateral ankle sprain
Chronic ankle instability
Lateral malleolar fracture
Peroneal tendon tear
Posterolateral ankle impingement
Osteochondral lesion of the talus
Sural nerve irritation
Treatment
General Principles
Treatment depends on whether the instability is:
Acute or chronic
and on the patient’s:
Activity level
Symptoms
Associated tendon injury
Acute Nonoperative Treatment
For a first-time acute injury, an initial period of nonoperative treatment may be attempted.
Options include:
Cast immobilization
Walking boot
Rest
Ice
NSAIDs when appropriate
Immobilization
Immobilization aims to allow the:
Superior peroneal retinaculum
to heal while preventing recurrent tendon displacement.
A period of immobilization is generally more effective for:
Acute injuries
than for established chronic instability.
Chronic Cases
Longstanding recurrent subluxation is less likely to respond to:
Casting or bracing alone.
Persistent painful instability often requires:
Operative stabilization.
Bracing and Taping
An ankle brace may reduce tendon excursion and improve symptoms.
Athletes may also use:
Taping
or
Lateral crescent/J-shaped pads
to help maintain the tendons behind the lateral malleolus.
These methods are primarily:
Symptom-control strategies
rather than definitive treatment for major structural instability.
Activity Modification
Reducing or avoiding the activity that provokes subluxation may help selected patients.
This is particularly reasonable when symptoms occur only with:
A specific high-demand movement or sport.
Physical Therapy
Physical therapy alone is generally insufficient to correct:
True mechanical peroneal tendon instability.
However, it is valuable for treating associated:
Weakness
Poor proprioception
Chronic ankle instability
and is especially important after surgery.
Rehabilitation Goals
Therapy should address:
Peroneal strengthening
Ankle range of motion
Balance
Proprioception
Neuromuscular control
Sport-specific function
Medication
NSAIDs may be used during the acute phase or rehabilitation to reduce:
Pain
and
Inflammation.
They do not correct the mechanical instability.
Surgery
Operative treatment is appropriate for:
Recurrent symptomatic subluxation
Failure of nonoperative treatment
Associated tendon tear
High-demand athletes requiring reliable stability
Competitive Athletes
High-level athletes may undergo earlier surgical treatment when recurrent instability is likely to interfere substantially with:
Return to play
and
Performance.
Surgical Principles
Surgery should address all contributing abnormalities rather than treating only the retinaculum.
Options include:
Superior peroneal retinacular repair
Peroneal tendon repair
Retromalleolar groove deepening
Correction of associated hindfoot malalignment when necessary
Retinacular Repair
When the superior peroneal retinaculum has been avulsed or torn, it can be:
Reattached or reconstructed
to restore the normal tunnel behind the fibula.
Groove Deepening
Patients with a:
Shallow or convex retromalleolar groove
may benefit from:
Fibular groove-deepening procedures
combined with retinacular repair.
This increases the bony containment of the tendons.
Tendon Repair
If a concomitant:
Peroneus brevis split tear
or other tendon lesion is present, treatment may include:
Debridement
Tubularization
or
Direct tendon repair.
Historical Procedures
Older procedures include:
Bone-block techniques
and
Tendon rerouting procedures beneath adjacent ligamentous structures.
These are now used less commonly because anatomic repair of the retinaculum and groove generally preserves more normal biomechanics.
Follow-Up
Postoperative rehabilitation is gradual.
Early treatment typically includes:
Immobilization
followed by progressive:
Range of motion
Strengthening
Proprioceptive training
Return to Activity
Patients may begin progressively increasing activity after approximately:
2–3 months, depending on healing and surgical technique.
Return to full competitive sports commonly requires approximately:
4–6 months, sometimes longer.
Prognosis
Chronic symptomatic instability has a relatively low likelihood of resolving permanently with conservative treatment alone.
Patients undergoing appropriate surgical stabilization generally achieve:
Good pain relief
Improved tendon stability
and
Return to athletic activity.
Complications
Potential surgical complications include:
Recurrent subluxation
Persistent pain
Peroneal tendon stiffness or adhesions
Sural nerve injury
Sural nerve entrapment
Sural neuroma
Recurrence
Recurrent instability may occur if:
Retinacular healing fails
Underlying groove abnormality is not corrected
Rehabilitation progresses too rapidly
Sural Nerve Injury
Because the sural nerve passes near the posterolateral ankle, surgery can result in:
Numbness
Neuropathic pain
or
Neuroma formation.
Patient Monitoring
Follow-up should assess:
Pain
Tendon stability
Range of motion
Peroneal strength
Ankle stability
Proprioception
Readiness to return to sport
Rehabilitation commonly requires renewed training in:
Ankle strengthening and proprioceptive control.
Key Principle
Peroneal tendon subluxation is dynamic instability of the peroneus longus and/or brevis tendons behind the lateral malleolus, usually caused by injury or insufficiency of the superior peroneal retinaculum.
The characteristic finding is:
Painful snapping with palpable or visible anterior displacement of the tendons over the distal fibula during ankle motion.
Acute injuries may be treated initially with immobilization, whereas:
Chronic, recurrent, or high-demand athletic cases frequently require surgical restoration of the retinaculum and associated anatomic abnormalities.